mitogen-activated protein kinase的意思|示意
促分裂原活化蛋白激酶
mitogen-activated protein kinase的网络常见释义
激酶 ...缺氧的保护性反应中起着重要作用。因此,探究某些药物治疗机制与HIF-1的关系,对开展脑卒中等严重威胁人类健康的疾病的有效治疗及预防具有重要意义。有丝分裂素激活蛋白激酶(mitogen-activated protein kinase,MAPK)是一组..
丝裂原活化蛋白激酶 丝裂原活化蛋白激酶(mitogen-activated protein kinase, MAPK)是调控细胞增殖和凋亡信号转导的关键酶。研究发现MAPK信号通路是参与ER基因组效应和非基因组效应的一...
丝裂原激活的蛋白激酶 丝裂原激活的蛋白激酶(mitogen-activated protein kinase,MAPK)是一组可以被多种细胞外信号激活的丝/苏氨酸激酶,其参与细胞的多种生物学行为,包括 ..
裂原激活蛋白激酶 丝裂原激活蛋白激酶(mitogen-activated protein kinase,MAPK)是多种细胞外刺激激活各种信号途径的交汇点，具有调节细胞基本生命活动的功能。
mitogen-activated protein kinase相关短语
1、 p38 mitogen-activated protein kinase 丝裂原活化蛋白激酶 ; 蛋白激酶 ; 活化蛋白激酶 ; 信号通路
2、 mitogen-activated protein kinase MAPK 丝裂原活化蛋白激酶 ; 裂原激活蛋白激酶 ; 促分裂原活化蛋白激酶 ; 蛋白激酶
3、 mitogen-activated protein kinase kinase 裂原活化蛋白激酶激酶 ; 蛋白激酶激酶 ; 蛋白激酶的激酶 ; 的激酶
mitogen-activated protein kinase相关例句
The extracellular signal-regulated kinase(ERK) is an important member of mitogen-activated protein kinase(MAPK) family.
细胞外信号调节激酶是丝裂原激活蛋白激酶家族的重要成员。
Objective to investigate the effect of mitogen-activated protein kinase (MAPK) on pathogenesis in myocardium hypertrophy of renal hypertension rats.
目的探讨丝裂素活化蛋白激酶(MAPK)在肾性高血压致心肌肥大中的作用。
Mitogen activated protein (MAP) kinase, also known as extracellular signal regulated kinase, or ERK, functions in neuronal signal transduction and participates in diverse development processes.
丝裂原激活的蛋白激酶又称细胞外信号调节激酶，具有神经元信号传导功能，参与多种发育过程。
Objective to investigate effects of PDGF on phenotypic change of glomerular mesangial cells (MC), and study intracellular mechanisms through mitogen-activated - protein kinase (MAPK) pathways.
目的研究血小板源生长因子(PDGF)对肾小球系膜细胞(MC)表型转化的影响，并探讨丝裂素活化蛋白激酶(MAPK)信号转导途径在其作用中的调控机制。
Mitogen-activated protein kinase pathway may closely associated with cell growth, differentiation and apoptosis, it may play an important role in cell death.
促有丝分裂原活化蛋白激酶（MAPK）信号传导途径与细胞生长、分化、凋亡等密切相关，在介导细胞凋亡过程中起着重要作用。
Mutations in GNA11 induced spontaneously metastasizing tumors in a mouse model and activated the mitogen-activated protein kinase pathway.
在小鼠模型中，GNA11突变诱导了自发转移性肿瘤，激活了丝裂原活化蛋白激酶途径。